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dc.contributor.authorGiacomin, Marina Mussoi-
dc.contributor.authorVilarinho, Gisele C.C.-
dc.contributor.authorCastro, Katia F.-
dc.contributor.authorFerreira, Márcio Soares-
dc.contributor.authorDuarte, Rafael Mendonça-
dc.contributor.authorWood, Chris M.-
dc.contributor.authorVal, Adalberto Luis-
dc.date.accessioned2020-06-15T21:37:18Z-
dc.date.available2020-06-15T21:37:18Z-
dc.date.issued2018-
dc.identifier.urihttps://repositorio.inpa.gov.br/handle/1/16919-
dc.description.abstractIncreasing anthropogenic activities in the Amazon have led to elevated metals in the aquatic environment. Since fish are the main source of animal protein for the Amazonian population, understanding metal bioaccumulation patterns and physiological impacts is of critical importance. Juvenile tambaqui, a local model species, were exposed to chronic dietary Cu (essential, 500 μg Cu/g food) and Cd (non-essential, 500 μg Cd/g food). Fish were sampled at 10–14, 18–20 and 33–36 days of exposure and the following parameters were analyzed: growth, voluntary food consumption, conversion efficiency, tissue-specific metal bioaccumulation, ammonia and urea-N excretion, O2 consumption, Pcrit, hypoxia tolerance, nitrogen quotient, major blood plasma ions and metabolites, gill and gut enzyme activities, and in vitro gut fluid transport. The results indicate no ionoregulatory impacts of either of the metal-contaminated diets at gill, gut, or plasma levels, and no differences in plasma cortisol or lactate. The Cd diet appeared to have suppressed feeding, though overall tank growth was not affected. Bioaccumulation of both metals was observed. Distinct tissue-specific and time-specific patterns were seen. Metal burdens in the edible white muscle remained low. Overall, physiological impacts of the Cu diet were minimal. However dietary Cd increased hypoxia tolerance, as evidenced by decreased Pcrit, increased time to loss of equilibrium, a lack of plasma glucose elevation, decreased plasma ethanol, and decreased NQ during hypoxia. Blood O2 transport characteristics (P50, Bohr coefficient, hemoglobin, hematocrit) were unaffected, suggesting that tissue level changes in metabolism accounted for the greater hypoxia tolerance in tambaqui fed with a Cd-contaminated diet. © 2018 Elsevier B.V.en
dc.language.isoenpt_BR
dc.relation.ispartofVolume 199, Pags. 30-45pt_BR
dc.rightsRestrito*
dc.subjectAlcoholen
dc.subjectAmmoniaen
dc.subjectCadmiumen
dc.subjectCopperen
dc.subjectGlucoseen
dc.subjectHemoglobinen
dc.subjectIntestine Enzymeen
dc.subjectIonen
dc.subjectNitrogenen
dc.subjectUreaen
dc.subjectAdenosine Triphosphatase (potassium Sodium)en
dc.subjectCadmiumen
dc.subjectCopperen
dc.subjectFish Proteinen
dc.subjectHydrocortisoneen
dc.subjectPotassiumen
dc.subjectSodiumen
dc.subjectBioaccumulationen
dc.subjectCadmiumen
dc.subjectCopperen
dc.subjectDieten
dc.subjectFood Consumptionen
dc.subjectHypoxiaen
dc.subjectMetabolismen
dc.subjectPhysiological Responseen
dc.subjectPollution Effecten
dc.subjectTeleosten
dc.subjectAlcohol Blood Levelen
dc.subjectAmazonasen
dc.subjectAnimals Cellen
dc.subjectAnimals Experimenten
dc.subjectAnimals Tissueen
dc.subjectAquatic Environmenten
dc.subjectBioaccumulationen
dc.subjectBiotransformationen
dc.subjectBody Growthen
dc.subjectBody Weight Gainen
dc.subjectColossoma Macropomumen
dc.subjectConcentration (parameters)en
dc.subjectControlled Studyen
dc.subjectEnzyme Activityen
dc.subjectFluid Transporten
dc.subjectFood Intakeen
dc.subjectGillen
dc.subjectGlucose Blood Levelen
dc.subjectGrowth Disorderen
dc.subjectHematocriten
dc.subjectHydrocortisone Blood Levelen
dc.subjectHypoxiaen
dc.subjectIn Vitro Studyen
dc.subjectIntestine Fluiden
dc.subjectLactate Blood Levelen
dc.subjectMetaboliteen
dc.subjectMetal Metabolismen
dc.subjectMineral Blood Levelen
dc.subjectMineral Intakeen
dc.subjectNonhumanen
dc.subjectOxygen Blood Levelen
dc.subjectOxygen Consumptionen
dc.subjectOxygen Tensionen
dc.subjectOxygen Transporten
dc.subjectPhysiologyen
dc.subjectPriority Journalen
dc.subjectTissue Levelen
dc.subjectTissue Specificityen
dc.subjectAnimalsen
dc.subjectBlooden
dc.subjectChemistryen
dc.subjectDieten
dc.subjectFishen
dc.subjectGastrointestinal Tracten
dc.subjectGrowth, Development And Agingen
dc.subjectKidneyen
dc.subjectLiveren
dc.subjectMetabolismen
dc.subjectPathologyen
dc.subjectWater Pollutanten
dc.subjectAnimalsiaen
dc.subjectColossoma Macropomumen
dc.subjectColossoma Marcopomumen
dc.subjectTeleosteien
dc.subjectAnimalen
dc.subjectCadmiumen
dc.subjectCopperen
dc.subjectDieten
dc.subjectFish Proteinsen
dc.subjectFishesen
dc.subjectGastrointestinal Tracten
dc.subjectGillsen
dc.subjectHydrocortisoneen
dc.subjectHypoxiaen
dc.subjectKidneyen
dc.subjectLiveren
dc.subjectOxygen Consumptionen
dc.subjectPotassiumen
dc.subjectSodiumen
dc.subjectSodium-potassium-exchanging Atpaseen
dc.subjectWater Pollutants, Chemicalen
dc.titlePhysiological impacts and bioaccumulation of dietary Cu and Cd in a model teleost: The Amazonian tambaqui (Colossoma macropomum)en
dc.typeArtigopt_BR
dc.identifier.doi10.1016/j.aquatox.2018.03.021-
dc.publisher.journalAquatic Toxicologypt_BR
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